Down regulation of CD11b and CD18 expression in atherosclerotic lesion-derived macrophages.
JL Gray, R Shankar - American Surgeon, 1995 - search.ebscohost.com
JL Gray, R Shankar
American Surgeon, 1995•search.ebscohost.comHypothesizes that monocyte/macrophages may not be able to exit atherosclerotic lesions
and reenter the circulation because of the reduced expression of CD11/CD18 integrins.
Strong cell surface expression of CD11b and CD18 in normal monocytes; CD11b and CD18
expression in atherosclerotic lesion-derived macrophages.
and reenter the circulation because of the reduced expression of CD11/CD18 integrins.
Strong cell surface expression of CD11b and CD18 in normal monocytes; CD11b and CD18
expression in atherosclerotic lesion-derived macrophages.
Abstract
Hypothesizes that monocyte/macrophages may not be able to exit atherosclerotic lesions and reenter the circulation because of the reduced expression of CD11/CD18 integrins. Strong cell surface expression of CD11b and CD18 in normal monocytes; CD11b and CD18 expression in atherosclerotic lesion-derived macrophages.
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