Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failure
G Kojda, D Harrison - Cardiovascular research, 1999 - academic.oup.com
G Kojda, D Harrison
Cardiovascular research, 1999•academic.oup.com1. Background: the concept of 'endothelial dysfunc- deficiency of either the substrate for the
enzyme NO tion' synthase or one of its critical co-factors. Alterations of endothelial cell
signaling may impair appropriate activation of the NO synthase in response to neurohumoral
or
enzyme NO tion' synthase or one of its critical co-factors. Alterations of endothelial cell
signaling may impair appropriate activation of the NO synthase in response to neurohumoral
or
1. Background: the concept of ‘endothelial dysfunc- deficiency of either the substrate for the enzyme NO tion’ synthase or one of its critical co-factors. Alterations of endothelial cell signaling may impair appropriate activation of the NO synthase in response to neurohumoral or
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